There are more than 140,000 miles of train track in the U.S., many of them laid across lonely stretches of land with nary a power line in sight. Thankfully, there?s a new gadget to harvest energy from vibrations generated in the track by passing trains to power signal lights and other track-side devices.
The energy harvester could save more than $10 million in trackside power costs in New York state alone, according to its inventor, Stony Brook University professor of mechanical engineering Lei Zuo. It would also reduce carbon dioxide emissions there by about 3,000 tons, he added.
The device converts the irregular up-and-down vibration of a train track to a unidirectional rotation of a generator. The concept is similar to other energy-harvesting devices such as the energy-generating backpack, kneebrace, speed bump?and pavers.?
The global market for energy harvesters hit $700 million in 2011 and could pass $4 billion by 2021, according to market research firm IDTechEx. Zuo and colleagues have licensed their Mechanical Motion Rectifier-based Railroad Energy Harvester to Electric Truck/Harvest NRG for commercialization.
? via Gizmag, Newsday, and SUNY?
John Roach is a contributing writer for NBC News Digital. To learn more about him, check out his website. For more of our Future of Technology series, watch the featured video below.
KABUL, Afghanistan (AP) ? A provincial official says a roadside bomb has killed 10 people and wounded eight in a remote part Afghanistan's southern Uruzgan province.
District Governor Nayamatullah Khaliqi said Thursday that the blast occurred late Wednesday and that most of the dead were women and children. He had no other details about the casualties from the blast near the village of Dehzak in the Dehra Wood district. All the victims were in a minivan that drove over a bomb.
Insurgents regularly plant roadside bomb to target NATO forces, but the devices often kill civilians. Roadside bombs have killed a total of 16 people since Tuesday.
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Being pre-approved can be a big part of that as well. If you have your home financing lined up ahead of time, it can make the purchase process go a lot smoother. We have a free e-book called "Demystifying the Mortgage Process", which can be helpful for new home buyers. You can access it through my profile page website.
ScienceDaily: Gene Newshttp://www.sciencedaily.com/news/health_medicine/genes/ Genes and Genetics News. Read today's medical research in genetics including what can damage genes, what can protect them, and more.en-usWed, 28 Nov 2012 17:06:54 ESTWed, 28 Nov 2012 17:06:54 EST60ScienceDaily: Gene Newshttp://www.sciencedaily.com/images/logosmall.gifhttp://www.sciencedaily.com/news/health_medicine/genes/ For more science articles, visit ScienceDaily.Double duty: Immune system regulator found to protect brain from effects of strokehttp://www.sciencedaily.com/releases/2012/11/121128143549.htm A small molecule known to regulate white blood cells has a surprising second role in protecting brain cells from the deleterious effects of stroke, researchers report. The molecule, microRNA-223, affects how cells respond to the temporary loss of blood supply brought on by stroke -- and thus the cells' likelihood of suffering permanent damage.Wed, 28 Nov 2012 14:35:35 ESThttp://www.sciencedaily.com/releases/2012/11/121128143549.htmScientists uncover a novel cooperative effort to stop cancer spreadhttp://www.sciencedaily.com/releases/2012/11/121128122041.htm Scientists have uncovered a group of what have been considered relatively minor regulators in the body that band together to suppress the spread of cancer from its primary site.Wed, 28 Nov 2012 12:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121128122041.htmChanges in nerve cells may contribute to the development of mental illnesshttp://www.sciencedaily.com/releases/2012/11/121128122035.htm Reduced production of myelin, a type of protective nerve fiber that is lost in diseases like multiple sclerosis, may also play a role in the development of mental illness, according to new research.Wed, 28 Nov 2012 12:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121128122035.htmFirst success of targeted therapy in most common genetic subtype of non-small cell lung cancerhttp://www.sciencedaily.com/releases/2012/11/121128121505.htm Chemotherapy and a new, targeted therapy work better in combination than chemotherapy alone in treating patients with the most common genetic subtype of lung cancer, new research suggests.Wed, 28 Nov 2012 12:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121128121505.htmImmune system could play a central role in age-related macular degenerationhttp://www.sciencedaily.com/releases/2012/11/121128093919.htm Changes in how genes in the immune system function may result in age-related macular degeneration (AMD), the leading cause of visual impairment in older adults. The findings are epigenetic in nature, meaning that the underlying DNA is normal but gene expression has been modified, likely by environmental factors, in an adverse way. Environmental factors associated with AMD include smoking, diet, and aging. This is the first epigenetic study revealing the molecular mechanisms for any eye disease.Wed, 28 Nov 2012 09:39:39 ESThttp://www.sciencedaily.com/releases/2012/11/121128093919.htmMany flame retardants in house dust at unsafe levels, study findshttp://www.sciencedaily.com/releases/2012/11/121128093810.htm In a new study of the largest number of flame retardants ever tested in homes, researchers found that most houses had levels of at least one flame retardant that exceeded a federal health guideline.Wed, 28 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121128093810.htmHow infidelity helps nieces and nephews: Men may share more genes with sisters' kids than cheating wife's kidshttp://www.sciencedaily.com/releases/2012/11/121127190021.htm A new study produced new mathematical support for a theory that explains why men in some cultures often feed and care for their sisters? children: where extramarital sex is common and accepted, a man?s genes are more likely to be passed on by their sister?s kids than by their wife?s kids.Tue, 27 Nov 2012 19:00:00 ESThttp://www.sciencedaily.com/releases/2012/11/121127190021.htmGene linked to respiratory distress in babieshttp://www.sciencedaily.com/releases/2012/11/121127130256.htm Some infants are more susceptible to potentially life-threatening breathing problems after birth, and rare, inherited DNA differences may explain why, according to new research.Tue, 27 Nov 2012 13:02:02 ESThttp://www.sciencedaily.com/releases/2012/11/121127130256.htmNew understanding of X chromosome inactivationhttp://www.sciencedaily.com/releases/2012/11/121127101534.htm Scientists have broadened our understanding of how cells regulate silencing of the X chromosome in a process known as X-inactivation.Tue, 27 Nov 2012 10:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121127101534.htmNew mechanism for cancer progression discoveredhttp://www.sciencedaily.com/releases/2012/11/121127094311.htm Researchers have discovered an alternative mechanism for activating rhe oncogene Ras that does not require mutation or hormonal stimulus.Tue, 27 Nov 2012 09:43:43 ESThttp://www.sciencedaily.com/releases/2012/11/121127094311.htmProtein injection points to muscular dystrophy treatmenthttp://www.sciencedaily.com/releases/2012/11/121127094248.htm Scientists have discovered that injecting a novel human protein into muscle affected by Duchenne muscular dystrophy significantly increases its size and strength, findings that could lead to a therapy akin to the use of insulin by diabetics.Tue, 27 Nov 2012 09:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121127094248.htmChemical 'switches' for neurodegenerative diseases discoveredhttp://www.sciencedaily.com/releases/2012/11/121127093951.htm Researchers have identified and ?switched off? a chemical chain that causes neurodegenerative diseases such as Huntington?s disease, amyotrophic lateral sclerosis and dementia. The findings could one day be of particular therapeutic benefit to Huntington?s disease patients.Tue, 27 Nov 2012 09:39:39 ESThttp://www.sciencedaily.com/releases/2012/11/121127093951.htmGene that causes tumor disorder linked to increased breast cancer riskhttp://www.sciencedaily.com/releases/2012/11/121127093855.htm New research showing a more than four-fold increase in the incidence of breast cancer in women with neurofibromatosis-1 (NF1) adds to growing evidence that women with this rare genetic disorder may benefit from early breast cancer screening with mammograms beginning at age 40, and manual breast exams as early as adolescence.Tue, 27 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121127093855.htmMetabolic protein launches sugar feast that nurtures brain tumorshttp://www.sciencedaily.com/releases/2012/11/121126164003.htm PKM2 slips into nucleus to promote cancer; potential biomarker and drug approach discovered.Mon, 26 Nov 2012 16:40:40 ESThttp://www.sciencedaily.com/releases/2012/11/121126164003.htmPossible new treatment for Ewing sarcomahttp://www.sciencedaily.com/releases/2012/11/121126142855.htm Discovery of a new drug with high potential to treat Ewing sarcoma, an often deadly cancer of children and young adults, and the previously unknown mechanism behind it, come hand-in-hand in a new study.Mon, 26 Nov 2012 14:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121126142855.htmSurvival gene may be key to controlling HIV and hepatitishttp://www.sciencedaily.com/releases/2012/11/121126131349.htm A newly discovered gene that is essential for embryo survival could also hold the key to treating and potentially controlling chronic infections such as HIV, hepatitis and tuberculosis. The gene, called Arih2, is fundamental to the function of the immune system -- making critical decisions about whether to switch on the immune response to an infection.Mon, 26 Nov 2012 13:13:13 ESThttp://www.sciencedaily.com/releases/2012/11/121126131349.htmMicrobial 'missing link' discovered after man impales hand on tree branchhttp://www.sciencedaily.com/releases/2012/11/121126110737.htm Two years ago, a 71-year-old Indiana man impaled his hand on a branch after cutting down a dead tree. The wound caused an infection that led scientists to discover a new bacterium and solve a mystery about how bacteria came to live inside insects.Mon, 26 Nov 2012 11:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121126110737.htmTransposable elements reveal a stem cell specific class of long noncoding RNAshttp://www.sciencedaily.com/releases/2012/11/121125192838.htm Over a decade after sequencing the human genome, it has now become clear that the genome is not mostly ?junk? as previously thought. In fact, the ENCODE project consortium of dozens of labs and petabytes of data have determined that these ?noncoding? regions house everything from disease trait loci to important regulatory signals, all the way through to new types of RNA-based genes.Sun, 25 Nov 2012 19:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121125192838.htmNew molecular culprit linked to breast cancer progressionhttp://www.sciencedaily.com/releases/2012/11/121124090511.htm Researchers have uncovered a protein ?partner? commonly used by breast cancer cells to unlock genes needed for spreading the disease around the body. A report on the discovery details how some tumors get the tools they need to metastasize.Sat, 24 Nov 2012 09:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121124090511.htmNew insights into virus proteome: Unknown proteins of the herpesvirus discoveredhttp://www.sciencedaily.com/releases/2012/11/121123092132.htm The genome encodes the complete information needed by an organism, including that required for protein production. Viruses, which are up to a thousand times smaller than human cells, have considerably smaller genomes. Using a type of herpesvirus as a model system scientists have shown that the genome of this virus contains much more information than previously assumed. The researchers identified several hundred novel proteins, many of which were surprisingly small.Fri, 23 Nov 2012 09:21:21 ESThttp://www.sciencedaily.com/releases/2012/11/121123092132.htmScientists describe elusive replication machinery of flu viruseshttp://www.sciencedaily.com/releases/2012/11/121122152928.htm Scientists have made a major advance in understanding how flu viruses replicate within infected cells. The researchers used cutting-edge molecular biology and electron-microscopy techniques to ?see? one of influenza?s essential protein complexes in unprecedented detail. The images generated in the study show flu virus proteins in the act of self-replication, highlighting the virus?s vulnerabilities that are sure to be of interest to drug developers.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152928.htmProtein folding: Look back on scientific advances made as result of 50-year old puzzlehttp://www.sciencedaily.com/releases/2012/11/121122152910.htm Fifty years after scientists first posed a question about protein folding, the search for answers has led to the creation of a full-fledged field of research that led to major advances in supercomputers, new materials and drug discovery, and shaped our understanding of the basic processes of life, including so-called "protein-folding diseases" such as Alzheimer's, Parkinson's and type II diabetes.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152910.htmStep forward in regenerating and repairing damaged nerve cellshttp://www.sciencedaily.com/releases/2012/11/121121145638.htm Researchers recently uncovered a nerve cell's internal clock, used during embryonic development. This breakthrough could lead to the development of new tools to repair and regenerate nerve cells following injuries to the central nervous system.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145638.htmArchitecture of rod sensory cilium disrupted by mutationhttp://www.sciencedaily.com/releases/2012/11/121121145621.htm Using a new technique called cryo-electron tomography, scientists have created a three-dimensional map that gives a better understanding of how the architecture of the rod sensory cilium (part of one type of photoreceptor in the eye) is changed by genetic mutation and how that affects its ability to transport proteins as part of the light-sensing process.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145621.htmAging: Scientists further unravel telomere biologyhttp://www.sciencedaily.com/releases/2012/11/121121130933.htm Researchers have resolved the structure of that allows a telomere-related protein, Cdc13, to form dimers in yeast. Mutations in this region of Cdc13 put the kibosh on the ability of telomerase and other proteins to maintain telomeres.Wed, 21 Nov 2012 13:09:09 ESThttp://www.sciencedaily.com/releases/2012/11/121121130933.htmDrug resistance biomarker could improve cancer treatmenthttp://www.sciencedaily.com/releases/2012/11/121121130811.htm Cancer therapies often have short-lived benefits due to the emergence of genetic mutations that cause drug resistance. A key gene that determines resistance to a range of cancer drugs has been reported in a new study. The study reveals a biomarker that can predict responses to cancer drugs and offers a strategy to treat drug-resistant tumors based on their genetic signature.Wed, 21 Nov 2012 13:08:08 ESThttp://www.sciencedaily.com/releases/2012/11/121121130811.htmGenome packaging: Key to breast cancer developementhttp://www.sciencedaily.com/releases/2012/11/121121130703.htm Two recent studies delve into the role of chromatin modifying enzymes and transcription factors in tumour cells. In one, it was found that the PARP1 enzyme activated by kinase CDK2 is necessary to induce the genes responsible for the proliferation of breast cancer cells in response to progesterone. In another, extensive work has been undertaken to identify those genes activated by the administration of progesterone in breast cancer, the sequences that can be recognized and how these genes are induced.Wed, 21 Nov 2012 13:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121121130703.htmShort DNA strands in genome may be key to understanding human cognition and diseaseshttp://www.sciencedaily.com/releases/2012/11/121121130643.htm Previously discarded, human-specific ?junk? DNA represents untapped resource in the study of diseases like Alzheimer?s and autism.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130643.htmBiomarking time: Methylome modifications offer new measure of our 'biological' agehttp://www.sciencedaily.com/releases/2012/11/121121130633.htm In a new study, researchers describe markers and a model that quantify how aging occurs at the level of genes and molecules, providing not just a more precise way to determine how old someone is, but also perhaps anticipate or treat ailments and diseases that come with the passage of time.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130633.htmKidney tumors have a mind of their ownhttp://www.sciencedaily.com/releases/2012/11/121121104552.htm New research has found there are several different ways that kidney tumors can achieve the same result -- namely, grow.Wed, 21 Nov 2012 10:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121121104552.htmMechanism to repair clumped proteins explainedhttp://www.sciencedaily.com/releases/2012/11/121121104416.htm Clumped proteins can be dissolved with the aid of cellular repair systems -- a process of critical importance for cell survival especially under conditions of stress. Researchers have now decrypted the fundamental mechanism for dissolving protein aggregates that involves specific molecular chaperones.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104416.htmNovel mechanism through which normal stromal cells become cancer-promoting stromal cells identifiedhttp://www.sciencedaily.com/releases/2012/11/121121104401.htm New understanding of molecular changes that convert harmless cells surrounding ovarian cancer cells into cells that promote tumor growth and metastasis provides potential new therapeutic targets for this deadly disease, according to new research.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104401.htmNew test for tuberculosis could improve treatment, prevent deaths in Southern Africahttp://www.sciencedaily.com/releases/2012/11/121120194932.htm A new rapid test for tuberculosis (TB) could substantially and cost-effectively reduce TB deaths and improve treatment in southern Africa -- a region where both HIV and tuberculosis are common.Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194932.htmEvolution of human intellect: Human-specific regulation of neuronal geneshttp://www.sciencedaily.com/releases/2012/11/121120194926.htm A new study has identified hundreds of small regions of the genome that appear to be uniquely regulated in human neurons. These regulatory differences distinguish us from other primates, including monkeys and apes, and as neurons are at the core of our unique cognitive abilities, these features may ultimately hold the key to our intellectual prowess (and also to our potential vulnerability to a wide range of 'human-specific' diseases from autism to Alzheimer's).Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194926.htmRibosome regulates viral protein synthesis, revealing potential therapeutic targethttp://www.sciencedaily.com/releases/2012/11/121120132906.htm Rather than target RNA viruses directly, aiming at the host cells they invade could hold promise, but any such strategy would have to be harmless to the host. Now, a surprising discovery made in ribosomes may point the way to fighting fatal viral infections such as rabies.Tue, 20 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121120132906.htmHow does antibiotic resistance spread? Scientists find answers in the nosehttp://www.sciencedaily.com/releases/2012/11/121120121835.htm Microbiologists studying bacterial colonization in mice have discovered how the very rapid and efficient spread of antibiotic resistance works in the respiratory pathogen, Streptococcus pneumoniae (also known as the pneumococcus). The team found that resistance stems from the transfer of DNA between bacterial strains in biofilms in the nasopharynx, the area just behind the nose.Tue, 20 Nov 2012 12:18:18 ESThttp://www.sciencedaily.com/releases/2012/11/121120121835.htmScientists identify inhibitor of myelin formation in central nervous systemhttp://www.sciencedaily.com/releases/2012/11/121120100155.htm Scientists have discovered another molecule that plays an important role in regulating myelin formation in the central nervous system. Myelin promotes the conduction of nerve cell impulses by forming a sheath around their projections, the so-called axons, at specific locations -- acting like the plastic insulation around a power cord.Tue, 20 Nov 2012 10:01:01 ESThttp://www.sciencedaily.com/releases/2012/11/121120100155.htm'Obese but happy gene' challenges the common perception of link between depression and obesityhttp://www.sciencedaily.com/releases/2012/11/121120084725.htm Researchers have discovered new genetic evidence about why some people are happier than others. The scientists have uncovered evidence that the gene FTO -- the major genetic contributor to obesity -- is associated with an eight per cent reduction in the risk of depression. In other words, it's not just an obesity gene but a "happy gene" as well.Tue, 20 Nov 2012 08:47:47 ESThttp://www.sciencedaily.com/releases/2012/11/121120084725.htmTelomere lengths predict life expectancy in the wild, research showshttp://www.sciencedaily.com/releases/2012/11/121119213144.htm Researchers have found that biological age and life expectancy can be predicted by measuring an individual's DNA. They studied the length of chromosome caps -- known as telomeres -- in a 320-strong wild population of Seychelles Warblers on a small isolated island.Mon, 19 Nov 2012 21:31:31 ESThttp://www.sciencedaily.com/releases/2012/11/121119213144.htmCancer: Some cells don't know when to stophttp://www.sciencedaily.com/releases/2012/11/121119171403.htm Certain mutated cells keep trying to replicate their DNA -- with disastrous results -- even after medications rob them of the raw materials to do so, according to new research.Mon, 19 Nov 2012 17:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121119171403.htmMultiple sclerosis ?immune exchange? between brain and blood is uncoveredhttp://www.sciencedaily.com/releases/2012/11/121119163301.htm DNA sequences obtained from a handful of patients with multiple sclerosis have revealed the existence of an ?immune exchange? that allows the disease-causing cells to move in and out of the brain.Mon, 19 Nov 2012 16:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121119163301.htmFruit fly studies guide investigators to molecular mechanism frequently misregulated in human cancershttp://www.sciencedaily.com/releases/2012/11/121119132056.htm Changes in how DNA interacts with histones ?- the proteins that package DNA ?- regulate many fundamental cell activities from stem cells maturing into a specific body cell type or blood cells becoming leukemic. These interactions are governed by a biochemical tug of war between repressors and activators, which chemically modify histones signaling them to clamp down tighter on DNA or move aside and allow a gene to be expressed.Mon, 19 Nov 2012 13:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121119132056.htm3-D light switch for the brain: Device may help treat Parkinson's, epilepsy; aid understanding of consciousnesshttp://www.sciencedaily.com/releases/2012/11/121119114249.htm A new tool for neuroscientists delivers a thousand pinpricks of light to individual neurons in the brain. The new 3-D "light switch", created by biologists and engineers, could one day be used as a neural prosthesis that could treat conditions such as Parkinson's and epilepsy by using gene therapy to turn individual brain cells on and off with light.Mon, 19 Nov 2012 11:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121119114249.htmNew factor of genetic susceptibility to Alzheimer's diseasehttp://www.sciencedaily.com/releases/2012/11/121119104944.htm A large-scale international study has just discovered a gene for susceptibility to a rare disease providing evidence of the heterogeneous aetiology of Alzheimer's disease.Mon, 19 Nov 2012 10:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121119104944.htmBlood cancer gene BCL6 identified as a key factor for differentiation of nerve cells of cerebral cortexhttp://www.sciencedaily.com/releases/2012/11/121119093848.htm The cerebral cortex is the most complex structure in our brain and the seat of consciousness, emotion, motor control and language. In order to fulfill these functions, it is composed of a diverse array of nerve cells, called cortical neurons, which are affected by many neurological and neuropsychiatric diseases. Researchers have opened new perspectives on brain development and stem cell neurobiology by discovering a gene called BCL6 as a key factor in the generation of cortical neurons during embryonic brain development.Mon, 19 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121119093848.htmMinority report: Insight into subtle genomic differences among our own cellshttp://www.sciencedaily.com/releases/2012/11/121118141530.htm Scientists have demonstrated that induced pluripotent stem cells -- the embryonic-stem-cell look-alikes whose discovery a few years ago won this year's Nobel Prize in medicine -- are not as genetically unstable as was thought.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141530.htmSkin cells reveal DNA's genetic mosaichttp://www.sciencedaily.com/releases/2012/11/121118141524.htm The prevailing wisdom has been that every cell in the body contains identical DNA. However, a new study of stem cells derived from the skin has found that genetic variations are widespread in the body's tissues, a finding with profound implications for genetic screening.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141524.htmLikely basis of birth defect causing premature skull closure in infants identifiedhttp://www.sciencedaily.com/releases/2012/11/121118141432.htm Geneticists, pediatricians, surgeons and epidemiologists have identified two areas of the human genome associated with the most common form of non-syndromic craniosynostosis premature closure of the bony plates of the skull.Sun, 18 Nov 2012 14:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121118141432.htmDNA packaging discovery reveals principles by which CRC mutations may cause cancerhttp://www.sciencedaily.com/releases/2012/11/121117184658.htm A new discovery concerning a fundamental understanding about how DNA works will produce a "180-degree change in focus" for researchers who study how gene packaging regulates gene activity, including genes that cause cancer and other diseases.Sat, 17 Nov 2012 18:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121117184658.htmHepatitis C treatment's side effects can now be studied in the labhttp://www.sciencedaily.com/releases/2012/11/121116161059.htm Adverse side effects of certain hepatitis C medications can now be replicated in the lab, thanks to a research team. The new method aids understanding of recent failures of hepatitis C antiviral drugs in some patients, and could help to identify medications that eliminate adverse effects. The findings may aid the development of safer and more effective treatments for hepatitis C and other pathogens such as SARS and West Nile virus.Fri, 16 Nov 2012 16:10:10 ESThttp://www.sciencedaily.com/releases/2012/11/121116161059.htmReconsidering cancer's bad guyhttp://www.sciencedaily.com/releases/2012/11/121116124644.htm Researchers have found that a protein, known for causing cancer cells to spread around the body, is also one of the molecules that trigger repair processes in the brain.Fri, 16 Nov 2012 12:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121116124644.htmGene distinguishes early birds from night owls and helps predict time of deathhttp://www.sciencedaily.com/releases/2012/11/121116124551.htm New research shows that a gene is responsible for a person's tendency to be an early riser or night owl -- and helps determine the time of day a person is most likely to die.Fri, 16 Nov 2012 12:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121116124551.htmClues to cause of kids' brain tumorshttp://www.sciencedaily.com/releases/2012/11/121116091226.htm Insights from a genetic condition that causes brain cancer are helping scientists better understand the most common type of brain tumor in children.Fri, 16 Nov 2012 09:12:12 ESThttp://www.sciencedaily.com/releases/2012/11/121116091226.htmArthritis study reveals why gender bias is all in the geneshttp://www.sciencedaily.com/releases/2012/11/121115210541.htm Researchers have pieced together new genetic clues to the arthritis puzzle in a study that brings potential treatments closer to reality and could also provide insights into why more women than men succumb to the disabling condition.Thu, 15 Nov 2012 21:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121115210541.htmClass of RNA molecules protects germ cells from damagehttp://www.sciencedaily.com/releases/2012/11/121115172255.htm Passing one's genes on to the next generation is a mark of evolutionary success. So it makes sense that the body would work to ensure that the genes the next generation inherits are exact replicas of the originals. Biologists have now identified one way the body does exactly that.Thu, 15 Nov 2012 17:22:22 ESThttp://www.sciencedaily.com/releases/2012/11/121115172255.htmQuick test speeds search for Alzheimer's drugs: Compound restores motor function and longevity to fruit flieshttp://www.sciencedaily.com/releases/2012/11/121115152655.htm Researchers report that an efficient, high-volume technique for testing potential drug treatments for Alzheimer's disease uncovered an organic compound that restored motor function and longevity to fruit flies with the disease.Thu, 15 Nov 2012 15:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115152655.htmProtein-making machinery can switch gears with a small structural change process; Implications for immunity and cancer therapyhttp://www.sciencedaily.com/releases/2012/11/121115133414.htm For the past several years, research has focused on the intricate actions of an ancient family of catalytic enzymes that play a key role in translation, the process of producing proteins. In a new study, scientists have shown that this enzyme can actually also work in another fundamental process in humans.Thu, 15 Nov 2012 13:34:34 ESThttp://www.sciencedaily.com/releases/2012/11/121115133414.htmPlant derivative, tanshinones, protects against sepsis, study suggestshttp://www.sciencedaily.com/releases/2012/11/121115133312.htm Researchers have discovered that tanshinones, which come from the plant Danshen and are highly valued in Chinese traditional medicine, protect against the life-threatening condition sepsis.Thu, 15 Nov 2012 13:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121115133312.htmStructure of enzyme topoisomerase II alpha unravelled providing basis for more accurate design of chemotherapeutic drugshttp://www.sciencedaily.com/releases/2012/11/121115132903.htm Medical researchers have for the first time described the structure of the active site core of topoisomerase II alpha, an important target for anti-cancer drugs. The type II topoisomerases are important enzymes that are involved in maintaining the structure of DNA and chromosome segregation during both replication and transcription of DNA. One of these enzymes, topoisomerase II alpha, is involved in the replication of DNA and cell proliferation, and is highly expressed in rapidly dividing cancer cells.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132903.htmNewly discovered enzyme important in the spreading of cancerhttp://www.sciencedaily.com/releases/2012/11/121115132901.htm Enzyme hunters at UiO have discovered the function of an enzyme that is important in the spreading of cancer. Cancer researchers now hope to inhibit the enzyme.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132901.htm
Surely they (Steve) didn't spend all that time designing the 4S. The 4S probably took them, like, 20 minutes to put together.
I tend to agree that the 5 isn't particularly "wowing", but by that account, looking at some of the iPhone upgrades, they weren't really that "wowing" either. However, the upgrades are just as significant (if not more) than were the upgrades from original to 3G, 3G to 3GS , or 4 to 4S.
Surfin? ain?t easy, and right now, you?re lost at sea. But don?t worry; simply pick an option from the list below, and you?ll be back out riding the waves of the Internet in no time.
Hit the ?back? button on your browser. It?s perfect for situations like this!
The plot of 'Zero Dark Thirty,' the movie by 'Hurt Locker' director Kathryn Bigelow, has been shrouded in secrecy. Check out the trailer to learn more.
By Sandy Schaefer,?Screen Rant / November 27, 2012
'Zero Dark Thirty' stars Joel Edgerton.
Jordan Strauss/Invision/AP
Enlarge
The new trailer for Hurt Locker screenwriter Mark Boal and director Kathryn Bigelow?s Zero Dark Thirty paints a tense, but vague, picture of their film ? which is about the decade-long manhunt for Osama bin Laden that culminated with Navy SEAL Team Six?s successful raid on the al-Qaeda head?s compound in Abbottabad, Pakistan.?We all know what the final outcome was, so the devil?s in the details.
Skip to next paragraph Screen Rant
Screen Rant had a humble start back in 2003 as a place to rant about some of the dumber stuff related to the movie industry. Since then, the site has grown to cover more and more TV and movie news (and not just the dumb stuff) along with sometimes controversial movie reviews. The goal at Screen Rant is to cover stories and review movies from a middle ground/average person perspective.
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Check out the trailer for Kathryn Bigelow's film about Osama bin Laden.
Bigelow and Boal have been playing things close to the chest on Zero Dark Thirty, so much so that the teaser trailer was technically the first official confirmation of the film?s cast. The new promo doesn?t shed light on how the duo are compressing so much information into a coherent narrative, though it does offer a quick sketch of the more central players in the film.
Judging by the new trailer, it appears Zero Dark Thirty partially centers on the efforts of an intelligence agent played by Oscar-nominee Jessica Chastain (Tree of Life, The Help), who uncovers vital information that could lead U.S. military forces to bin Laden?s secret location. However, we also glimpse important participants like Mark Strong and Kyle Chandler as fellow CIA agents, Joel and Nash Edgerton as members of SEAL Team Six, and James Gandolfini ? who EW has confirmed is playing the current Secretary of Defense, Leon Panetta.
Overall, according to Bigelow (via?EW), there are over 100 speakings roles in Zero Dark Thirty, including ?teams of operatives, from [Department of Defense], CIA, Navy SEALs, et al. that intersect with foreign nationals and enemy combatants.? So, although Chastain and Strong get the most screen time in the trailer, this is very much going to be an ensemble piece.
Now, as we?ve discussed in the past, there?s no doubt that Bigelow and Boal have their work cut out for them. It doesn?t matter how exhaustively they?ve researched the history behind the bin Laden manhunt ? or how few liberties they take with the facts ? Zero Dark Thirty is going to take a pounding from all corners of the political spectrum, with contradictory accusations about its alleged socio-political biases and fabrications flying in every direction?(thank goodness the U.S. election will be over by that point).
However, few filmmakers know how to create pulse-pounding suspense and nerve-wracking thrills like Bigelow. Plus, she and Boal demonstrated they know how to create enthralling cinema together with the Best Picture-winning Hurt Locker. So, on those grounds, this movie?s easily worth recommending for anyone who?s just interested in an intense, well-acted viewing experience.
Sandy Schaefer blogs at Screen Rant.
The Christian Science Monitor has assembled a diverse group of music, film, and television bloggers. Our guest bloggers are not employed or directed by The Monitor and the views expressed are the bloggers' own and they are responsible for the content of their blogs. To contact us about a blogger, click here.
Apple hopes to take more prisoners in its patent war against arch rival Samsung, asking a judge to add six of the Korean company’s flagship phones and tablets to the list of gadgets infringing upon its patents. Apple contends it ...
US Auto Repair and Maintenance Industry Explored in New Market Report
The US auto?mo?tive repair and main?te?nance ser?vices indus?try is expected to grow at a low rate over the next two years. More con?sumers replac?ing their old cars after post?pon?ing new car pur?chases dur?ing the reces?sion may con?tribute to slow indus?try growth in the near term, accord?ing to Research and Mar?kets ?Auto?mo?tive Repair & Main?te?nance Ser?vices? report.
The indus?try includes about 160,000 estab?lish?ments with com?bined annual rev?enue of about $80 bil?lion. Major com?pa?nies include Bridge?stone, Jiffy Lube, Meineke (a divi?sion of Dri?ven Brands), Midas, Monro Muf?fler Brake, and Safe?lite Group. Large com?pa?nies can max?i?mize use of expen?sive diag?nos?tic equip?ment and have advan?tages in pur?chas?ing, dis?tri?b?u?tion, and mar?ket?ing. Small com?pa?nies can com?pete effec?tively by pro?vid?ing supe?rior cus?tomer ser?vice or offer?ing spe?cial?ized services.
The indus?try is highly frag?mented: the 50 largest com?pa?nies gen?er?ate less than 10 per?cent of rev?enue, accord?ing to the mar?ket report. Com?pe?ti?tion includes other venues that pro?vide auto?mo?tive ser?vices, includ?ing some gas sta?tions, car deal?er?ships, and branches of chain stores, like Sears and?Kmart.
Visualization meditation may be the single most powerful self-improvement tool you have. Use it to relax, relieve pain, create new thought habits, empower yourself, achieve goals, heal your body, clear up past emotional issues and more.
If you can imagine it, you can live it!
Why is visualization so powerful? Because the mind does not operate using words. Self-talk is very important but the mind instantly translates and processes every word that is thought, heard or read into an image.
?Purple notebook.?
You read a bunch of black squiggles on a white page, but your mind painted a picture. It?s like this with any thought you have.
Unfortunately, most of the images we habitually hold in our heads are not positive, empowering or healing. They are typically not loving, peaceful or happy (if they were, you would be living your ideal healthy, happy and fulfilled life). Unless you are an exceptionally self-aware and self-controlled person, your thoughts will center around:
Worry about the future
Regrets about the past
Problems
That?s not to say that positive-thinking people have their heads in the sand and are in denial about reality! You want to be aware of your problems, but when you focus on the problem (the fact that the problem exists) you cannot be focused on the solution.
Visualization meditation helps you become solution-oriented rather than problem-oriented. That means, when you think about the future, you will do so with a focus on how you want it. When you think about the past, you will see it as a chapter in your personal history book, but you won?t remain caught up in it (wishing you could undo, redo, relive or fix anything that happened). You?ll see the past as a series of essential lessons.
And, you will immediately set your mind to work in creating a solution.
Use the Silva Method visualization techniques to harness the creative energy of your imagination. The Mental Screen exercise is a way to paint a mental picture of what you want that is not based on ?what is.?
The best time to do visualization meditation is just before you go to sleep. When you direct your imagination to what you want, you give your mind a very clear command. When you do this just before you go to sleep, you will set the wheels of manifesting in motion! Your mind will have all night to ponder the situation, weigh the options, come up with solutions? and when you do this consistently (to make your mind aware that this is really important and you want this!) you will become hyper-aware of your surroundings. You?ll start noticing that what you need is right there in front of you, and now your mind has brought it to your attention. You?ll notice resources. People. Events. Opportunities. And when you take action on these, you?ll quickly move away from the problem to a solution.
How To Practice Visualization Meditation
Sit comfortably (avoid lying in bed because you might fall asleep before you?ve had a chance to clarify and enhance your mental images), eyes closed.
Focus on your breath and allow your body to relax. Your thoughts will slow down as you enter the alert-yet-relaxed alpha brainwave state.
Think about a problem you?re having, but keep your emotions out of it as much as possible. Be very clinical about it: ?this is the problem.?
Fire up your imagination and visualize the end result you want. Don?t give a moment?s thought to how you?re going to achieve this. Forget all the intermediate stuff. That?s your mind?s job and it will do its job if you give it the right command! Always visualize in the present tense, as though what you want is already yours.
The next step is to create positive emotions about your desired end result. Feel happy, elated, relieved, excited and confident that what you want is coming to you and that you are enjoying it (present tense).
Think of it this way:
You go to a restaurant because you have a problem: you?re hungry. Your mind comes up with mental images of various dishes as they are described on the menu. You order your dish, and you visualize yourself eating and enjoying it! You don?t care how your dish is going to be prepared, you only care THAT it will come to you (hopefully soon, before you fill up on too much bread). You sit and enjoy pleasant conversation with your dining partner, all the while confidently anticipating that your dish will arrive, and holding in your mind the feeling of satisfaction of eating your food.
So take that same attitude with any problem you have. Give the visual command, and then release it. Allow your mind to get to work and take immediate action on the information and inspirations you receive, and let go of worry. It?s being handled!
Another way of using visualization meditation is to release negativity. This can be particularly useful when you?re holding onto a grudge. Many of us hold grudges ? and these are destructive energies, a total waste of mental space, and a huge negative energetic burden. Use a technique called the ?pink bubble? (or another color if you prefer). Place an unwanted thought into the bubble, release it and watch it float away from you.
Acknowledge the thought and the accompanying feeling, and use your imagination to place the thought/feeling into a bubble where it can?t get to you and you can?t get to it. You don?t own it anymore. You don?t have to live with the burden anymore. You can still remember the event, but without the emotional attachment to it.
The pink bubble technique takes practice, but you?ll find that it?s a great way to help release the past.
And anytime you find yourself worrying, use visualization meditation to see the result you want and give it increasing levels of mental energy until it becomes a ?no options? command to your mind.
Geometries presented by Chinese scholars for all possible space-time kinematics and their relationsPublic release date: 26-Nov-2012 [ | E-mail | Share ]
Contact: Yan Bei yanbei@scichina.org Science China Press
The possible kinematics and their corresponding geometries were once regarded as an already-solved problem. The de Sitter relativity research group formed by researchers from Chinese Academy of Sciences, Tsinghua University, and Beijing Normal University, restudied the problem and showed that additional, previously unknown realizations exist of possible kinematical algebras, each of which has so(3) isotropy and a ten-generators symmetry group. They presented these geometries corresponding to all these realizations and provided a classification in an article, entitled "Geometries for Possible Kinematics", published in the 2012 10th issue of SCIENCE CHINA [1] .
In the 1960s Bacry and Lvy-Leblond established connections among eleven kinematical algebras of eight types. Each kinematical algebra was supposed to possess (i) an so(3) isotropy, (ii) parity and time-reversal automorphisms, and (iii) a non-compact one-dimensional subgroup generated by each boost. For a long time, it was widely accepted that the accounting for all kinematical algebras satisfying those three conditions had been exhausted. Two years ago, the de Sitter relativity research group showed by using linear combinations of generators that there are 24 kinematical algebras if the third condition is relaxed [2], and all of those kinematical algebras are subalgebras of a 4-dimensional "inertial motion algebra".
In Ref. [1], it was shown that, with the exception of two static algebras, the 22 possible kinematical algebras with so(3) isotropy can be obtained by the Inn-Wigner contraction from the Riemann, Lobachevsky, de Sitter, and anti-de Sitter algebras (r, l, d), respectively. The existence of more possible kinematical algebras than obtained by Bacry and Lvy-Leblond arises from taking different realizations of the generators and then performing the contraction under two opposite limits. For example, it is well known that the de Sitter and anti-de Sitter algebras contract to the Poincar algebra (p) when a certain length parameter tends to infinity. What was overlooked was that when the length parameter tends to zero, [3] these algebras contract to other realizations of the Poincar algebra, called the second Poincar algebras and denoted p2, for brevity. Although these are isomorphic, p and p2 have very different geometrical significance. The 22 possible kinematical algebras are related, as depicted in figure 1.
More importantly, a kinematic action should be established on a suitable geometry so that the geometry is invariant under the transformations generated by the kinematical algebra. In Ref. [1], the geometries for all 22 kinematical algebras were presented using the contraction technique. As arranged in figure 2, there are 45 different 4-dimensional geometries in total. Each geometry is defined on a portion of a 4-dimensional real projective manifold. Among these geometries, some are non-degenerate like the de Sitter and Minkowski geometries, but most are degenerate as for example for the Galilei and Carroll geometries. In these geometries, including the degenerate ones, some have Lorentzian signature that may serve as space-time geometries, and some have Euclidean signature that may serve as Euclidean versions of space-time. Moreover, there are geometries which have a (+, +, -, -)-signature and are interpreted as double-time geometries. Many of the geometries are related by the transformation t 1/(n2t). When this transformation is regarded as a coordinate transformation, these geometries are not independent from the view of differential geometry.
The explicit geometric structures show that the requirement that transformations generated by boosts in any given direction form a noncompact subgroup does not guarantee a geometry having Lorentz-like signature. Some geometries satisfying that requirement possess Euclidean signature, whereas others violating that requirement possess Lorentz-like signature. In addition, before the geometry is presented, the isotropy (rotational invariance) of the space is expressed by an so(3) subalgebra. However, although many geometries are invariant under transformations generated by so(3), they might not have spatial isotropy with respect to each point in the manifold.
Therefore, the requirements suitable in identifying genuine possible kinematics have been revised to the following: (1) space is isotropic with respect to any point in the manifold; (2) space-reflection and time-reversal are automorphisms of the kinematical group; and (3) the geometry has Lorentz-like signature. According to those requirements, the genuine possible kinematics from the viewpoint of differential geometry correspond to three relativistic geometries, three absolute-time geometries and three absolute-space geometries (see Table 1).
###
1. HUANG CG, TIAN Y, WU XN, XU Z and ZHOU B, Geometries for possible kinematics, SCI CHINA Phys. Mech. Astron. 2012, 55(11), doi: 10.1007/s11433-012-4788-4.
2. GUO HY, HUANG CG, WU HT and ZHOU B, SCIENCE CHINA Phys. Mech. Astron. 2012, Vol 53(4) 591
3. HUANG CG, In Proceedings of the 9th Asia-Pacific International Conference. Singapore ed Luo J et al World Scientific Publishing, 2010. 118; HUANG CG, TIAN Y,WU XN, et al. Chin Phys Lett, 2012, 29: 040303; Commun Theor Phys, 2012, 57: 553.
[ | E-mail | Share ]
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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Geometries presented by Chinese scholars for all possible space-time kinematics and their relationsPublic release date: 26-Nov-2012 [ | E-mail | Share ]
Contact: Yan Bei yanbei@scichina.org Science China Press
The possible kinematics and their corresponding geometries were once regarded as an already-solved problem. The de Sitter relativity research group formed by researchers from Chinese Academy of Sciences, Tsinghua University, and Beijing Normal University, restudied the problem and showed that additional, previously unknown realizations exist of possible kinematical algebras, each of which has so(3) isotropy and a ten-generators symmetry group. They presented these geometries corresponding to all these realizations and provided a classification in an article, entitled "Geometries for Possible Kinematics", published in the 2012 10th issue of SCIENCE CHINA [1] .
In the 1960s Bacry and Lvy-Leblond established connections among eleven kinematical algebras of eight types. Each kinematical algebra was supposed to possess (i) an so(3) isotropy, (ii) parity and time-reversal automorphisms, and (iii) a non-compact one-dimensional subgroup generated by each boost. For a long time, it was widely accepted that the accounting for all kinematical algebras satisfying those three conditions had been exhausted. Two years ago, the de Sitter relativity research group showed by using linear combinations of generators that there are 24 kinematical algebras if the third condition is relaxed [2], and all of those kinematical algebras are subalgebras of a 4-dimensional "inertial motion algebra".
In Ref. [1], it was shown that, with the exception of two static algebras, the 22 possible kinematical algebras with so(3) isotropy can be obtained by the Inn-Wigner contraction from the Riemann, Lobachevsky, de Sitter, and anti-de Sitter algebras (r, l, d), respectively. The existence of more possible kinematical algebras than obtained by Bacry and Lvy-Leblond arises from taking different realizations of the generators and then performing the contraction under two opposite limits. For example, it is well known that the de Sitter and anti-de Sitter algebras contract to the Poincar algebra (p) when a certain length parameter tends to infinity. What was overlooked was that when the length parameter tends to zero, [3] these algebras contract to other realizations of the Poincar algebra, called the second Poincar algebras and denoted p2, for brevity. Although these are isomorphic, p and p2 have very different geometrical significance. The 22 possible kinematical algebras are related, as depicted in figure 1.
More importantly, a kinematic action should be established on a suitable geometry so that the geometry is invariant under the transformations generated by the kinematical algebra. In Ref. [1], the geometries for all 22 kinematical algebras were presented using the contraction technique. As arranged in figure 2, there are 45 different 4-dimensional geometries in total. Each geometry is defined on a portion of a 4-dimensional real projective manifold. Among these geometries, some are non-degenerate like the de Sitter and Minkowski geometries, but most are degenerate as for example for the Galilei and Carroll geometries. In these geometries, including the degenerate ones, some have Lorentzian signature that may serve as space-time geometries, and some have Euclidean signature that may serve as Euclidean versions of space-time. Moreover, there are geometries which have a (+, +, -, -)-signature and are interpreted as double-time geometries. Many of the geometries are related by the transformation t 1/(n2t). When this transformation is regarded as a coordinate transformation, these geometries are not independent from the view of differential geometry.
The explicit geometric structures show that the requirement that transformations generated by boosts in any given direction form a noncompact subgroup does not guarantee a geometry having Lorentz-like signature. Some geometries satisfying that requirement possess Euclidean signature, whereas others violating that requirement possess Lorentz-like signature. In addition, before the geometry is presented, the isotropy (rotational invariance) of the space is expressed by an so(3) subalgebra. However, although many geometries are invariant under transformations generated by so(3), they might not have spatial isotropy with respect to each point in the manifold.
Therefore, the requirements suitable in identifying genuine possible kinematics have been revised to the following: (1) space is isotropic with respect to any point in the manifold; (2) space-reflection and time-reversal are automorphisms of the kinematical group; and (3) the geometry has Lorentz-like signature. According to those requirements, the genuine possible kinematics from the viewpoint of differential geometry correspond to three relativistic geometries, three absolute-time geometries and three absolute-space geometries (see Table 1).
###
1. HUANG CG, TIAN Y, WU XN, XU Z and ZHOU B, Geometries for possible kinematics, SCI CHINA Phys. Mech. Astron. 2012, 55(11), doi: 10.1007/s11433-012-4788-4.
2. GUO HY, HUANG CG, WU HT and ZHOU B, SCIENCE CHINA Phys. Mech. Astron. 2012, Vol 53(4) 591
3. HUANG CG, In Proceedings of the 9th Asia-Pacific International Conference. Singapore ed Luo J et al World Scientific Publishing, 2010. 118; HUANG CG, TIAN Y,WU XN, et al. Chin Phys Lett, 2012, 29: 040303; Commun Theor Phys, 2012, 57: 553.
[ | E-mail | Share ]
?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Credit cards are convenient only if used responsibly. One must never forget that one should spend as much as he or she can afford to pay back at the end of the month. It is over spending that brings on trouble for card users. People often spend mindlessly and then repaying the kredi kart? taksitlendirme becomes problematic which, ultimately, results in the accumulation of huge debts.
You should treat your credit amount as a borrowed sum that should be repaid soon. And for this, you need to make sure to repay the kredi kart? borcu taksitlendirme at the end of the current billing cycle.
If you too, like several other people, have got trapped in the maze of debts, the best way to get over it is to lower it as soon as possible. And the best place to go to for help is a credit-counseling company. They are well-experienced in consolidating such debts as fast as it possibly can be done. There are many ways in which you can lower your interest rates but not all the ways are suitable to everybody. The credit card company will analyse your current financial condition and will suggest you a way suitable for you. S Debt consolidatoin is a way many people choose to decrease credit card interest. It involves the payment of all your dues with decreased monthly payment. It helps you in a way that it lets you to make a single payment instead of numerous ones for each of the cards used by you every month. Also when you pay in this way, the companies can reduce your high interest rates over time, especially if you are required to use your credit card in the future. But you will have to keep asking your creditor to do so, otherwise this might not be done. The lower your balance is in your credit card, the better are the chances for you to get low interest rates on your card.
Debt settlement is also a popular advice that the credit-counseling companies offer. This is usually meant for people who are lagging behind on their debts and have also missed a few kredi kart? borcu taksitlendirme. This, however, affects your credit card rating, so it is better to go for other options before you try this. This method involves negotiation between the credit card counseller and the compnany that had issued the card where the former tries to get you the permission to pay a lumpsum amount for the outstanding debt, instead of kredi kart? taksitlendirme. This thus involves one payment which is considerably less than what you actually owe.
Another way is the home equity line of credit under which loans are given on the basis of how much your home is worth than your present debt. The loan is usually provided against the difference in the values.
These programs can help you reduce your interest rates and also eliminate your dues in much lesser time and with much less money. The earlier you decide to seek help the better it is for you.
Kredi Kart? Taksitlendirme is the name who writes this article for people as a free, informative and helpful publication.
WASHINGTON (AP) ? Sorry, fellas, but President Barack Obama's re-election makes it official: Women can overrule men at the ballot box.
For the first time in research dating to 1952, a presidential candidate whom men chose decisively ? Republican Mitt Romney ? lost. More women voted for the other guy.
It's surprising it didn't happen sooner because women have been voting in larger numbers than men for almost three decades, exit polls show.
But men, who make up less than half the U.S. population, always have exercised power greater than their numbers and they aren't about to stop now.
When it comes to elections, males as a group are more influential because they show less party loyalty than women, who skew Democratic.
Despite all the focus on candidates courting Hispanics or the working class, men are the nation's ultimate swing voters; they're why Republican George W. Bush became president and Republican John McCain didn't.
Their move away from Obama this year expanded the voting "gender gap." It wasn't enough to determine the outcome, but came close.
So presidential hopefuls staring into the gender gap in 2016 might want to look beyond the usual controversies over "women's issues" such as abortion or the polling fads such as "Wal-Mart moms." Maybe it's time to pause and consider the fickle male. Maybe it's time to ask, "What do men want?"
In the voting booth, that is.
"I don't think we fully understand it yet," political scientist Christina Wolbrecht of the University of Notre Dame said about why men and women vote differently. But she said plenty of research on elections going back to the 1950s indicates it's not because of issues such as equal pay, birth control coverage in health plans or Romney's awkward reference to "binders full of women."
Paul Kellstedt has some ideas. A Texas A&M associate professor of political science, Kellstedt studies what American men and women want from their government and how that shifts over time.
Like Wolbrecht, he noted that the sexes aren't that different, at least when it comes to the issues.
Studies have found that the opinions that separate liberals and conservatives, even on issues such as abortion, don't divide the sexes much. Men and women are about as likely to fall on either side of those debates, and millions of each happily line up with each political party.
But there has been a consistent thread of disagreement for decades over what role the government should play. It's not a big gap, but it is statistically significant, about 4 percentage points or 5 points in many studies, Kellstedt said. As a group, women tend to like bigger government with more health and welfare programs; men lean toward smaller government that spends less, except on the military.
Sort of the social safety net versus rugged individualism. Or Obama versus Romney.
There are lots of possible reasons the genders see this differently.
Besides women's traditional role as family nurturers, they also live longer than men and so are more likely to rely on Social Security and Medicare. Women are more likely to be poor. They're more likely to be single parents struggling to pay for child care, education and medical bills. Men may feel many social programs are expensive and won't benefit them.
"Women tend to believe that government has a role to play, that it should be a partner in their life," said Democratic pollster Celinda Lake. "Men tend to think it's been a good day when the government hasn't done anything bad to you."
When the nation as a whole drifts to the left or right on the big government-small government debate, the gap between men and women fluctuates. Men and women shift their views in the same direction, Kellstedt said, but men as a group tend to change their minds faster and move their views farther.
"The variation among men's opinions is larger," he said. "The flighty, moody ones are the men, not the women."
He said this difference of opinion on the role of government isn't big enough to entirely explain the larger gender gap in voting, however. "It's a little bit of a puzzle."
Women as a group voted Democratic in the past six presidential races, from 1992 through 2012, according to exit polls. The last time they decisively supported a Republican was Ronald Reagan's re-election in 1984. The Reagan years were when Americans first began taking note of the "gender gap," as women's rights groups emphasized that female support for Reagan in 1980 was narrow while male voters overwhelmingly endorsed him.
Men lean Republican but play the field. In the past six presidential races, men voted Republican three times, Democratic twice (including barely supporting Obama in 2008), and essentially split their vote in the 1996 Bill Clinton-Bob Dole race, exit polls show.
This year Obama campaigned on giving a leg up to those needing education, health care or job training. Romney talked about shrinking government, except for the military, and said overgrown social programs were creating a culture of dependency. Their arguments fit the long-running fissure of the gender gap.
"Women stuck with Obama," said Karen Kaufmann, a University of Maryland associate professor who studies the gender gap. "We didn't see a lot of movement from women. The movement was really men going back to the Republican Party."
Women's support for Obama dropped just 1 percentage point from 2008; they voted for him by 55 percent to 44 percent this time. Men's support for Obama dropped 4 points, flipping them to Romney's side, by a 52-45 margin. Women were 10 percentage points more likely to vote for Obama than men were, according to the survey of voters at the polls conducted for The Associated Press and television networks.
Gallup polling has tracked the gender gap since 1952. Gallup says this year's gender divide was 20 percentage points, the largest ever using its method of calculation.
The gender gap isn't just a white thing. It exists even among minorities that vote overwhelmingly Democratic. Obama got 96 percent of black women's votes, but 87 percent of black men's, compared with 76 percent of Hispanic women and 65 percent of Hispanic men, according to the exit poll.
"We group together this white male vote and sort of put that in the Republican ledger and we don't talk enough about all the various subgroups that fit within men and the multiple issues and currents that determine how they're going to vote," said sociologist Donald Levy, director of the Siena College's research institute.
"The Democrats aren't succeeding with some of these folks," Levy said. The Democratic Party needs to figure out why, he said, the same way "the Republicans are doing some soul-searching about how they can appeal to women."
___
Follow Connie Cass on Twitter: http://www.twitter.com/ConnieCass
I can't think of them off the top of my head right now. But I think you see where I'm getting at.
Visualization meditation may be the single most powerful self-improvement tool you have. Use it to relax, relieve pain, create new thought habits, empower yourself, achieve goals, heal your body, clear up past emotional issues and more.
The best time to do visualization meditation is just before you go to sleep. When you direct your imagination to what you want, you give your mind a very clear command. When you do this just before you go to sleep, you will set the wheels of manifesting in motion! Your mind will have all night to ponder the situation, weigh the options, come up with solutions? and when you do this consistently (to make your mind aware that this is really important and you want this!) you will become hyper-aware of your surroundings. You?ll start noticing that what you need is right there in front of you, and now your mind has brought it to your attention. You?ll notice resources. People. Events. Opportunities. And when you take action on these, you?ll quickly move away from the problem to a solution.
Another way of using visualization meditation is to release negativity. This can be particularly useful when you?re holding onto a grudge. Many of us hold grudges ? and these are destructive energies, a total waste of mental space, and a huge negative energetic burden. Use a technique called the ?pink bubble? (or another color if you prefer). Place an unwanted thought into the bubble, release it and watch it float away from you.
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Public release date: 26-Nov-2012
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